Effect of liver regeneration on malignant hepatic tumors

doi:10.3748/wjg.v20.i43.16167

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Nov 21, 2014 (publication date) through Apr 18, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Nov 21, 2014; 20(43): 16167-16177
Published online Nov 21, 2014. doi: 10.3748/wjg.v20.i43.16167

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Figure 2 Molecule-mediated signal pathways in the propagation of liver cancer during liver regeneration. NF-κB and JAK/STAT3 pathways, Phosphatidylinositol-3 kinase (PI3K)/AKT pathway, Ras/Raf/Mek/Erk pathway, TGF-β/SMAD pathway, WNT/β-catenin pathway and activation of nuclear transcription are implicated in this process. EGFR: Epidermal growth factor/transforming growth factor α receptor; FGFR: Fibroblast growth factor receptor; HGFR: Hepatocyte growth factor receptor; PDGFR: Platelet-derived growth factor receptor; TGFβ: Transforming growth factor beta receptor; VEGFR: Vascular endothelial growth factor receptor.

Citation: Shi JH, Line PD. Effect of liver regeneration on malignant hepatic tumors. World J Gastroenterol 2014; 20(43): 16167-16177
URL: https://www.wjgnet.com/1007-9327/full/v20/i43/16167.htm
DOI: https://dx.doi.org/10.3748/wjg.v20.i43.16167