Copyright
©The Author(s) 2003.
World J Gastroenterol. Apr 15, 2003; 9(4): 788-790
Published online Apr 15, 2003. doi: 10.3748/wjg.v9.i4.788
Published online Apr 15, 2003. doi: 10.3748/wjg.v9.i4.788
Figure 1 Three days after the induction of gastric ulcers, Tie-2 expression in the endothelial cells of new capillaries was en-hanced in the rhIL-11-treated rats (A) in comparison to the untreated control animals (B).
Figure 2 Western blots demonstrating Tie-2 expression.
RhIL-11 treatment increased Tie-2 expression significantly 3 and 5 d after the induction of gastric ulcers compared with un-treated control rats.
Figure 3 RT-PCR analysis of Tie-2 mRNA expression in ulcer-ative gastric tissues, using the specific primer pairs predicted to amplify Tie-2 (317 bp) and β-actin (313 bp).
Lanes 1-4: rhIL-11-treated rats on days 3 (lane 2) and 5 (lane 4) and the untreated control rats on day 3 (lane 1) and 5 (lane 3).
- Citation: Wen CY, Ito M, Wang H, Chen LD, Xu ZM, Matsuu M, Shichijo K, Nakayama T, Nakashima M, Sekine I. IL-11 up-regulates Tie-2 expression during the healing of gastric ulcers in rats. World J Gastroenterol 2003; 9(4): 788-790
- URL: https://www.wjgnet.com/1007-9327/full/v9/i4/788.htm
- DOI: https://dx.doi.org/10.3748/wjg.v9.i4.788