Causal role of Helicobacter pylori infection in gastric cancer

doi:10.3748/wjg.v12.i2.181

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Jan 14, 2006 (publication date) through Apr 23, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Jan 14, 2006; 12(2): 181-186
Published online Jan 14, 2006. doi: 10.3748/wjg.v12.i2.181

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Figure 1 Deregulation of SHP-2 tyrosine phosphatase by the H pylori virulence factor CagA. CagA is delivered into gastric epithelial cells by the bacterial type IV secretion system. CagA deregulates the SHP2 oncoprotein. CagA is noted for its variation, particularly at the SHP2-binding site.

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Figure 2 Multistep model for the progression of gastric cancer.

Citation: Ando T, Goto Y, Maeda O, Watanabe O, Ishiguro K, Goto H. Causal role of Helicobacter pylori infection in gastric cancer. World J Gastroenterol 2006; 12(2): 181-186
URL: https://www.wjgnet.com/1007-9327/full/v12/i2/181.htm
DOI: https://dx.doi.org/10.3748/wjg.v12.i2.181