Polyunsaturated fatty acids and DNA methylation in colorectal cancer

doi:10.12998/wjcc.v7.i24.4172

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Dec 26, 2019 (publication date) through May 24, 2024

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World Journal of Clinical Cases

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2307-8960

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Clin Cases. Dec 26, 2019; 7(24): 4172-4185
Published online Dec 26, 2019. doi: 10.12998/wjcc.v7.i24.4172

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Figure 1 Four general mechanisms by which polyunsaturated fatty acids can induce their antineoplastic effects in colorectal cancer. PUFA: Polyunsaturated fatty acid; CRC: Colorectal cancer; COX-2; Cyclooxygenase-2: Mt: Mitochondria.

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Figure 2 A model of the possible mechanisms by which polyunsaturated fatty acids can alter DNA methylation in colorectal cancer. DNMTs: DNA methyltransferases; DHA: Docosahexaenoic acid; PE-DHA: Phosphatidylethanolamine-DHA; PC-DHA: Phosphatidylcholine-DHA; PUFA: Poly unsaturated fatty acid; COX-2: Cyclooxygenase-2; PGE2: Prostaglandin E2; PPARs: Peroxisome proliferator-activated receptors; RXR: Retinoid X receptor; UHRF1: Ubiquitin-like protein containing PHD and RING finger domains 1; miRNAs: MicroRNAs; α-KG: α-ketoglutarate; TET: Ten-eleven translocation.

Citation: Moradi Sarabi M, Mohammadrezaei Khorramabadi R, Zare Z, Eftekhar E. Polyunsaturated fatty acids and DNA methylation in colorectal cancer. World J Clin Cases 2019; 7(24): 4172-4185
URL: https://www.wjgnet.com/2307-8960/full/v7/i24/4172.htm
DOI: https://dx.doi.org/10.12998/wjcc.v7.i24.4172