Hyperhomocysteinemia as a potential contributor of colorectal cancer development in inflammatory bowel diseases: A review

doi:10.3748/wjg.v21.i4.1081

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Jan 28, 2015 (publication date) through Apr 25, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Jan 28, 2015; 21(4): 1081-1090
Published online Jan 28, 2015. doi: 10.3748/wjg.v21.i4.1081

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Figure 1 Metabolism of homocysteine[7]. dUMP: Desoxyuridine monophosphate; dTMP: Desoxytimidine monophosphste; THF: Tetrahydrofolate; DHF: Dihydrofolate; 5-MTHF: 5-methyltetrahydrofolate; 5,10-MTHF: 5,10-methyltetrahydrofolate; 5,10 MTHFR: 5,10- methyltetrahydrofolate reductase; MS: Metionin synthase; MSR: Metionin synthase reductase; B12: Vitamin B12; SAM: S-adenosylmethionine; SAH: S-adenosylhomocysteine; CBS: Cystathionine β-synthase; GCT: γ-cystathionase; B6: Vitamin B6.

Citation: Keshteli AH, Baracos VE, Madsen KL. Hyperhomocysteinemia as a potential contributor of colorectal cancer development in inflammatory bowel diseases: A review. World J Gastroenterol 2015; 21(4): 1081-1090
URL: https://www.wjgnet.com/1007-9327/full/v21/i4/1081.htm
DOI: https://dx.doi.org/10.3748/wjg.v21.i4.1081