Immune reaction and colorectal cancer: Friends or foes?

doi:10.3748/wjg.v20.i35.12407

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Sep 21, 2014 (publication date) through Apr 19, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Sep 21, 2014; 20(35): 12407-12419
Published online Sep 21, 2014. doi: 10.3748/wjg.v20.i35.12407

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Figure 1 Model proposed by Rakoff-Nahoum et al[30]. Competent TLR4/TLR2/Myd88 pathway is physiologically activated by commensal micro-organisms and maintains epithelial homeostasis (A). Deficiency in TLR4/TLR2/Myd88 signal determines increased epithelial vulnerability and bacterial translocation with chronic colitis and cancer development (B). IECs: Intestinal epithelial cells; TLR: Toll-like receptor; CRC: Colorectal cancer; IL-6: Interleukin-6; MyD88: Myeloiddifferentiationfactor88.

Citation: Formica V, Cereda V, Nardecchia A, Tesauro M, Roselli M. Immune reaction and colorectal cancer: Friends or foes? World J Gastroenterol 2014; 20(35): 12407-12419
URL: https://www.wjgnet.com/1007-9327/full/v20/i35/12407.htm
DOI: https://dx.doi.org/10.3748/wjg.v20.i35.12407