Basic Research
Copyright ©2006 Baishideng Publishing Group Co.
World J Gastroenterol. Aug 14, 2006; 12(30): 4859-4865
Published online Aug 14, 2006. doi: 10.3748/wjg.v12.i30.4859
Figure 1
Figure 1 Effects of ALR on proliferation of Kupffer cells and hepatocytes. A: DNA replication; B: BrdU-positive nuclei. KC: Kupffer cells; HC: Hepatocytes; KC to HC: Hepatocytes conditioned by Kupffer cells. aP < 0.05 vs control, n = 3.
Figure 2
Figure 2 BrdU immunohistochemistry. The nuclei of BrdU-positive cells were stained brown. A: Kupffer cells; B: Hepatocytes; C: Hepatocytes conditioned by Kupffer cells (× 100).
Figure 3
Figure 3 3H-leucine incorporation assay. A: Kupffer cells and associated medium; B: Hepatocytes and associated medium; C: Hepatocytes conditioned by Kupffer cells and associated medium. aP < 0.05 vs control, n = 3.
Figure 4
Figure 4 125I-rrALR binding assay. A: Saturation cures of 125I-rrALR binding to its receptor on Kupffer cells; B: Scatchard plot of the ALR receptor on Kupffer cells; C: Binding of ALR to cultured hepatocytes (mean ± SD, n = 3).