Case Report
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World J Gastroenterol. Dec 21, 2014; 20(47): 18038-18043
Published online Dec 21, 2014. doi: 10.3748/wjg.v20.i47.18038
Eosinophilic esophagitis in patients with esophageal atresia and chronic dysphagia
Sirvart Kassabian, Virginia Baez-Socorro, Thomas Sferra, Reinaldo Garcia
Sirvart Kassabian, Reinaldo Garcia, Department of Pediatric Gastroenterology, Nutrition and Hepatology, Akron Children’s Hospital, Akron, OH 44308, United States
Virginia Baez-Socorro, Thomas Sferra, Division of Pediatric Gastroenterology, Nutrition and Hepatology, Rainbow Babies and Children’s Hospital, Cleveland, OH 44106, United States
Author contributions: Kassabian S and Baez-Socorro V wrote the original manuscript; Sferra T helped with editing; Garcia R had seen multiple patients with this association and guided the writing of the manuscript; all the authors contributed to the manuscript.
Correspondence to: Sirvart Kassabian, MD, Department of Pediatric Gastroenterology, Nutrition and Hepatology, Akron Children’s Hospital, One Perkins Square, Akron, OH 44308, United States. skassabian@chmca.org
Telephone: +1-330-5434488 Fax: +1-330-5435060
Received: March 13, 2014
Revised: July 22, 2014
Accepted: September 18, 2014
Published online: December 21, 2014
Core Tip

Core tip: Dysphagia is frequently seen in patients with repaired esophageal atresia (EA). It has been attributed to recurrent strictures, poor esophageal motility and persistent gastroesophageal reflux disease. Anastomitic strictures are common after repair of a gap that is greater than 2.5 cm contributing to this complication. The pathophysiology of later onset dysphagia is not well defined. Eosinophilic esophagitis (EoE) has been reported to play a role in the reoccurrence of strictures in patients with EA. It is very likely that if these patients are treated for EoE early in the course of the disease, stricture formation might be prevented.