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©2014 Baishideng Publishing Group Inc. All rights reserved.
World J Gastroenterol. Dec 7, 2014; 20(45): 17196-17205
Published online Dec 7, 2014. doi: 10.3748/wjg.v20.i45.17196
Published online Dec 7, 2014. doi: 10.3748/wjg.v20.i45.17196
Relationship between the exocrine and endocrine pancreas after acute pancreatitis
Stephanie L M Das, James I C Kennedy, Anthony R J Phillips, John A Windsor, Maxim S Petrov, Department of Surgery, University of Auckland, Auckland 1142, New Zealand
Rinki Murphy, Department of Medicine, University of Auckland, Auckland 1142, New Zealand
Author contributions: Das SLM acquired data and drafted the manuscript; Kennedy JIC acquired data, carried out data analysis, and drafted the manuscript; Murphy R, Phillips ARJ and Windsor JA contributed to interpretation of data and critically reviewed the manuscript; and Petrov MS conceived and supervised the study.
Correspondence to: Maxim S Petrov, MD, MPH, PhD, Department of Surgery, University of Auckland, Private Bag 92019, Auckland 1142, New Zealand. max.petrov@gmail.com
Telephone: +64-9-9232776 Fax: +64-9-3779656
Received: March 19, 2014
Revised: July 9, 2014
Accepted: July 29, 2014
Published online: December 7, 2014
Revised: July 9, 2014
Accepted: July 29, 2014
Published online: December 7, 2014
Core Tip
Core tip: Diabetes mellitus and pancreatic exocrine insufficiency are common after acute pancreatitis. Concomitant pancreatic exocrine insufficiency occurs in 40% of patients with prediabetes or diabetes and its prevalence decreases with time. Purposefully designed clinical studies are required to elucidate the pathogenesis of pancreatogenic diabetes.