Metabolic liver disease of obesity and role of adipose tissue in the pathogenesis of nonalcoholic fatty liver disease

doi:10.3748/wjg.v13.i26.3540

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379

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Jul 14, 2007 (publication date) through May 10, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Jul 14, 2007; 13(26): 3540-3553
Published online Jul 14, 2007. doi: 10.3748/wjg.v13.i26.3540

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Figure 1 Lipid metabolism in liver. All steps indicated by + are stimulated by insulin. Insulin suppresses the secretion of VLDL and the β-oxidation of fatty acids. Thus hyperinsulinemia in the setting of insulin resistance favors TAG accumulation in liver. TAG: triglycerides; VLDL: very low density lipoprotein; FA: fatty acid; NEFA: nonesterified fatty acids; DNL: de novo lipogenesis; ATP: adenosine triphosphate.

Citation: Qureshi K, Abrams GA. Metabolic liver disease of obesity and role of adipose tissue in the pathogenesis of nonalcoholic fatty liver disease. World J Gastroenterol 2007; 13(26): 3540-3553
URL: https://www.wjgnet.com/1007-9327/full/v13/i26/3540.htm
DOI: https://dx.doi.org/10.3748/wjg.v13.i26.3540