Aging and uremia: Is there cellular and molecular crossover?

doi:10.5527/wjn.v4.i1.19

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Feb 6, 2015 (publication date) through Apr 19, 2024

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World Journal of Nephrology

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2220-6124

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Nephrol. Feb 6, 2015; 4(1): 19-30
Published online Feb 6, 2015. doi: 10.5527/wjn.v4.i1.19

Table 1 Events common to aging and uremia covered by this review

Aging	Uremia
TGF-β↑	TGF-β↑
Autophagy ↓	Autophagy ↓
Apoptosis ↑ (muscle)	Apoptosis ↑
Senescence ↑	Senescence ↑
Telomere shortening ↑	Telomere shortening ↑
Stem cell exhaustion ↑	Stem cell exhaustion ↑
Klotho ↓	Klotho ↓
AGEs ↑	AGEs ↑
Mitochondrial dysfunction ↑	Mitochondrial dysfunction ↑

TGF-β: Transforming growth factor beta; AGEs: Advanced glycation end products.

Citation: White WE, Yaqoob MM, Harwood SM. Aging and uremia: Is there cellular and molecular crossover? World J Nephrol 2015; 4(1): 19-30
URL: https://www.wjgnet.com/2220-6124/full/v4/i1/19.htm
DOI: https://dx.doi.org/10.5527/wjn.v4.i1.19