Copyright ©The Author(s) 2015.
World J Nephrol. May 6, 2015; 4(2): 185-195
Published online May 6, 2015. doi: 10.5527/wjn.v4.i2.185
Figure 1
Figure 1 Haematuria-induced kidney injury in tubular cells. Hb: Haemoglobin; Bb: Bilirubin; Bv-red: Biliverdin reductase; CO: Carbon monoxide; Fe: Iron; HO-1: Heme oxygenase 1; MCP: Monocyte chemoattractant protein; NF-κb: Nuclear factor kappa b; TGF-β: Transforming growth factor beta; TNF-α: Tumour necrosis factor alpha.
Figure 2
Figure 2 Glomerular filtration barrier structure and red blood cell egression leading to haematuria. CL: Capillary lumen; BC: Bowman’s capsule; E: Endothelial cell; GBM: Glomerular basement membrane; Gly: Glycosaminoglicans; M: Mesangium; P: Podocyte; RBC: Red blood cell; SD: Slit diaphragm; SP: Subpodocyte space; TC: Tubular cell; US: Urinary space.