Pathogenesis of glomerular haematuria

doi:10.5527/wjn.v4.i2.185

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World Journal of Nephrology

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2220-6124

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Nephrol. May 6, 2015; 4(2): 185-195
Published online May 6, 2015. doi: 10.5527/wjn.v4.i2.185

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Figure 1 Haematuria-induced kidney injury in tubular cells. Hb: Haemoglobin; Bb: Bilirubin; Bv-red: Biliverdin reductase; CO: Carbon monoxide; Fe: Iron; HO-1: Heme oxygenase 1; MCP: Monocyte chemoattractant protein; NF-κb: Nuclear factor kappa b; TGF-β: Transforming growth factor beta; TNF-α: Tumour necrosis factor alpha.

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Figure 2 Glomerular filtration barrier structure and red blood cell egression leading to haematuria. CL: Capillary lumen; BC: Bowman’s capsule; E: Endothelial cell; GBM: Glomerular basement membrane; Gly: Glycosaminoglicans; M: Mesangium; P: Podocyte; RBC: Red blood cell; SD: Slit diaphragm; SP: Subpodocyte space; TC: Tubular cell; US: Urinary space.

Citation: Yuste C, Gutierrez E, Sevillano AM, Rubio-Navarro A, Amaro-Villalobos JM, Ortiz A, Egido J, Praga M, Moreno JA. Pathogenesis of glomerular haematuria. World J Nephrol 2015; 4(2): 185-195
URL: https://www.wjgnet.com/2220-6124/full/v4/i2/185.htm
DOI: https://dx.doi.org/10.5527/wjn.v4.i2.185