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Cited by in CrossRef
For: Lai ZF, Chen YZ. Evidence, hypotheses and significance of MAP kinase TNNI3K interacting with its partners. World J Hypertens 2012; 2(2): 22-28 [DOI: 10.5494/wjh.v2.i2.22]
URL: https://www.wjgnet.com/2220-3168/full/v2/i2/22.htm
Number Citing Articles
1
Pavithra K. Balasubramanian, Anand Balupuri, Swapnil P. Bhujbal, Seung Joo Cho. 3D-QSAR Assisted Design of Novel 7-Deazapurine Derivatives as TNNI3K Kinase Inhibitors Using Molecular Docking and Molecular Dynamics SimulationLetters in Drug Design & Discovery 2020; 17(2): 155 doi: 10.2174/1570180816666190110121300
2
Shafaq Ramzan, Stephanie Tennstedt, Muhammad Tariq, Sheraz Khan, Hafiza Noor Ul Ayan, Aamir Ali, Matthias Munz, Holger Thiele, Asad Aslam Korejo, Abdul Razzaq Mughal, Syed Zahid Jamal, Peter Nürnberg, Shahid Mahmood Baig, Jeanette Erdmann, Ilyas Ahmad. A Novel Missense Mutation in TNNI3K Causes Recessively Inherited Cardiac Conduction Disease in a Consanguineous Pakistani FamilyGenes 2021; 12(8): 1282 doi: 10.3390/genes12081282
3
Yanwei Xi, Christina Honeywell, Dapeng Zhang, Jeremy Schwartzentruber, Chandree L. Beaulieu, Martine Tetreault, Taila Hartley, Jennifer Marton, Silvia M. Vidal, Jacek Majewski, L. Aravind, Michael Gollob, Kym M. Boycott, Robert M. Gow. Whole exome sequencing identifies the TNNI3K gene as a cause of familial conduction system disease and congenital junctional ectopic tachycardiaInternational Journal of Cardiology 2015; 185: 114 doi: 10.1016/j.ijcard.2015.03.130