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Copyright ©The Author(s) 2015.
World J Diabetes. Apr 15, 2015; 6(3): 489-499
Published online Apr 15, 2015. doi: 10.4239/wjd.v6.i3.489
Table 1 Types of diabetic maculopathy[19]
FocalLocalized edema Lipid exudates Intraretinal hemorrhages Focal hyperfluorescence in late fluorescein angiography
Clinically significant without foveal thickening (non-center-involving) (sight-threatening)Edema within 500 μm around the foveola Exudates within 500 μm around the foveola accompanied by edema Edema ≥ 1 optic-disk diameter within one optic-disk diameter around the foveola
Clinically significant with foveal thickening (center-involving)Ill-defined edema, which may be cystoid Exudates Intraretinal hemorrhages Origin of leakage often not clearly identifiable by fluorescein angiography
TractionalDue to vitreous traction to the fovea Thickened posterior hyaloid membrane OCT visualizes vitreal traction
Ischemic maculopathy (occlusion of the perifoveal capillaries)Loss of vision without any clearly visible cause on fundoscopy Fluorescein angiography needed for diagnosis Difficult to diagnose by fundoscopy only Edema may be present or absent
Table 2 Recommended timing of retinal examinations in patients with type 2 diabetes[6]
Patient characteristicsTiming of retinal examination
Initial diagnosis of type 2 diabetesSoon
No diabetic retinopathyOnce a year
Presence of symptoms such asDuring the next few days
Loss of vision
New difficulties during reading
Altered color perception
New, moving dark spots in the eye
Presence of diabetic retinopathyDepending on the severity of retinopathy, e.g., every 3-6 mo