Editorial
Copyright ©2010 Baishideng Publishing Group Co.
World J Gastrointest Oncol. Oct 15, 2010; 2(10): 369-379
Published online Oct 15, 2010. doi: 10.4251/wjgo.v2.i10.369
Figure 1
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Figure 1 Steps in Helicobacter pylori-related gastric cancer. H. pylori: Helicobacter pylori; NOD1: Nucleotide-binding oligomerization domain protein 1; PTPN11: Protein-tyrosine phosphatase non receptor-type 11; RUNX3: Runt-related gene 3; cagPAI: cag pathogenicity island.
Figure 2
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Figure 2 Changes in the gastric epithelial cells due to the activation of Helicobacter pylori cag pathogenicity island-related molecules. H. pylori: Helicobacter pylori; iE-DAP: γ-D-glutamyl-meso-diaminopimelic acid; iNOS: Inducible nitric oxide synthase; NF-κB, Nuclear factor κB; NOD1: Nucleotide-binding oligomerization domain protein 1; PAR1: Partitioning-defective-1; SHP-2: Src homology 2 domain-containing protein tyrosine phosphatase-2; cagPAI: cag pathogenicity island.
Figure 3
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Figure 3 Signal pathways from Helicobacter pylori to cytokine gene expression through innate immunity. H. pylori: Helicobacter pylori; IKK: Inhibitory κB kinase; iE-DAP: γ-D-glutamyl-meso-diaminopimelic acid; IRAK: Interleukin 1 receptor-associated kinase; MyD88: Myeloid differentiation factor 88; NF-κB, Nuclear factor κB; NOD1: Nucleotide-binding oligomerization domain protein 1; SOCS: Suppressor of cytokine signaling; TOLLIP: Toll-interacting protein; TIRAP: TIR domain-containing adaptor protein.