Coexpression of receptor-tyrosine-kinases in gastric adenocarcinoma-a rationale for a molecular targeting strategy?

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Jul 14, 2007 (publication date) through Apr 26, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Jul 14, 2007; 13(26): 3605-3609
Published online Jul 14, 2007. doi: 10.3748/wjg.v13.i26.3605

Table 1 Antibody characteristics

Target	Antibody	Dilution	Incubation
VEGFR1	Flt-1 (C-17)	1:100	4 h	Santa Cruz, Biotechnology, CA, USA
VEGFR3	Flt-4 (C20)	1:200	2 h	Santa Cruz, Biotechnology, CA, USA
PDGFRα	PDGFRα (C-20)	1:200	2 h	Santa Cruz, Biotechnology, CA, USA
PDGFRβ	PDGFRβ (28 E1)	1:200	2 h	Cell Signalling Technology, MA, USA
EGFR1	AM207-5ME	1:1	1 h	BioGenex, CA, USA

Citation: Drescher D, Moehler M, Gockel I, Frerichs K, Müller A, Dünschede F, Borschitz T, Biesterfeld S, Holtmann M, Wehler T, Teufel A, Herzer K, Fischer T, Berger MR, Junginger T, Galle PR, Schimanski CC. Coexpression of receptor-tyrosine-kinases in gastric adenocarcinoma-a rationale for a molecular targeting strategy? World J Gastroenterol 2007; 13(26): 3605-3609
URL: https://www.wjgnet.com/1007-9327/full/v13/i26/3605.htm
DOI: https://dx.doi.org/10.3748/wjg.v13.i26.3605