ABH and Lewis antigen distributions in blood, saliva and gastric mucosa and H pylori infection in gastric ulcer patients

Table 1 Distribution of ABO and Lewis phenotypes in erythrocytes and saliva of gastric ulcer patients and controls

Lewiserythrocytes	Saliva		Secretor	LewisphenotypesBlood/Salive	ABO Phenotypes
					A1		A2		B		AB		O
	Lea	Leb			I	II	I	II	I	II	I	II	I	II
Le (a+ b-)	+	+	S	Concordant	1	2	1	-	-	1	1	-	2	-
Le (a- b+)	+	+	S	Concordant	1	7	2	2	1	7	-	1	12	20
Le (a- b-)	-	-	S	Concordant	-	2	-	-	-	-	-	-	2	-
	+	+	S	Discordant	3	-	1	1	4	-	-	-	7	7
	-	+	S	Discordant	1	-	-	-	1	-	-	-	2	-
Total					6	11	4	3	6	8	1	1	25	27

I = gastric ulcer patients, II = control, S = secretor status

Table 2 Distribution of ABO and Lewis blood group phenotypes in relation to serological diagnosis of H pylori infection in ulcer patients and controls

Blood group	H pylori infection
	Patientsa			Controlsb
phenotypes	N	Positive	Negative	N	Positive	Negative
A2 Le(a-b+) ou O Le(a-b+)	27	26	1	30	25	5
A1/AB/B/Le(a-b+) ou O Le(a-b-)	15	12	3	20	5	15
Total	42	38	4	50	30	20

^aP > 0.05 vs bacterial density, lymphocytic and granulocytic infiltration degree;

^bP < 0.01 vs polymorph nuclear activity.