Clinical analysis of 36 cases of hepatopulmonary syndrome

doi:10.3748/wjg.v4.iSuppl2.128

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Oct 15, 1998 (publication date) through Apr 24, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Oct 15, 1998; 4(Suppl2): 128-128
Published online Oct 15, 1998. doi: 10.3748/wjg.v4.iSuppl2.128

Clinical analysis of 36 cases of hepatopulmonary syndrome

Hong Wei, Yu-Xuan Wang

Hong Wei, Yu-Xuan Wang, Department of Gastroenterology, Hainan Provincial People’s Hospital, Haikou 570311, Hainan Province, China

Author contributions: All authors contributed equally to the work.

Correspondence to: Dr. Hong Wei, Department of Gastroenterology, Hainan Provincial People’s Hospital, Haikou 570311, Hainan Province, China

Received: July 10, 1998
Revised: August 11, 1998
Accepted: September 7, 1998
Published online: October 15, 1998

Abstract

AIM: To analyse the causes and pathogenesis of Hepatopulmonary Syndrome (HPS) in patients with liver cirrhosis.

METHODS: Femoral artery blood samples of 102 cases with liver cirrhosis were taken right after their admission to the hospital to detect the PaO₂ and SaO₂.

RESULTS: PaO₂ and SaO₂ in patients with HPS were markedly lower than that without HPS (P < 0.01); PaO₂ and SaO₂ in patients with a liver function of child C were markedly lower than that of child A (P < 0. 01); HPS incidence in patients with liver function of child C was markedly higher than that with child A (P < 0.01); The average PaO₂ and SaO₂ in patients with pylephlebectasis ( > 1.4 in diameter) were markedly lower than that without pylephlebectasis (P < 0.01); HPS incidence in patients with pylephlebectasis was markedly higher than that without pylephlebectasis (P < 0.001); PaO₂ and SaO₂ in patients with cutaneous spider nevi were markedly lower than that without cutaneous spider nevi (P < 0.05); HPS incidence in patients with cutaneous spider nevi was markedly higher than that without cutaneous spider nevi (P < 0.001).

CONCLUSION: HPS might occure in any stage of patients with liver cirrhosis. The more severely the liver function was damaged, the more chances there were to suffer from HPS, and the more severe the symptoms would be.

Key Words: Liver cirrhosis, Lung diseases/physiopathology, Lung diseases/diagnosis, Syndrome

Citation: Wei H, Wang YX. Clinical analysis of 36 cases of hepatopulmonary syndrome. World J Gastroenterol 1998; 4(Suppl2): 128-128
URL: https://www.wjgnet.com/1007-9327/full/v4/iSuppl2/128.htm
DOI: https://dx.doi.org/10.3748/wjg.v4.iSuppl2.128

Footnotes

E- Editor: Li RF