Basic Research
Copyright ©The Author(s) 2003.
World J Gastroenterol. Jun 15, 2003; 9(6): 1292-1295
Published online Jun 15, 2003. doi: 10.3748/wjg.v9.i6.1292
Figure 1
Figure 1 Loss of normal lobular architecture, some had pseudo lobule formation in the CCl4 intoxicated groups. HE × 100.
Figure 2
Figure 2 Steatosis was not severe and fibrosis was not so pronounced, without any pseudo -lobule formation in the Ganyanping-treated groups. HE × 100.
Figure 3
Figure 3 The numbers of hepatic stellate cells and collagen fibrils increased in Disse's space and hepatocellular degeneration were frequently seen in the CCl4 intoxicated groups. × 4000.
Figure 4
Figure 4 Most of hepatocytes showed basically normal ultrastructure and a few lipid droplets were found in the cytoplasm in the Ganyanping-treated groups. × 4000.
Figure 5
Figure 5 Hepatic parenchyma separated by the rough, red-stained fibrotic septa in the CCl4 intoxicated groups. VG × 200.
Figure 6
Figure 6 Collagen fibrosis was less pronounced in the Ganyanping-treated groups. VG × 200.
Figure 7
Figure 7 Increase in the activities of MAO(+++) in the CCl4 intoxicated groups. × 75.
Figure 8
Figure 8 Decrease in the activities of MAO in the Ganyanping-treated groups (++). × 75.