Roles of Fas signaling pathway in vitamin E succinate-induced apoptosis in human gastric cancer SGC-7901 cells

Figure 1 SGC-7901 cells stained with DAPI. A: UT control; B: VES at 20 mg·L^-1

Figure 2 The expression of Fas protein in SGC-7901 cells follow-ing treatment of VES for 24 h. Lane1: Molecular weight marker; Lane2: UT control; Lane3: succinate; Lane4: vitamin E; Lane5: VES at 5 mg·L^-1; Lane6: VES at 10 mg·L^-1; Lane7: VES at 20 mg·L^-1.

Figure 3 The expression of FADD protein in SGC-7901 cells fol-lowing treatment of VES for 24 h. Lane1: Molecular weight marker; Lane2: VES at 20 mg·L^-1; Lane3: VES at 10 mg·L^-1; Lane4: VES at 5 mg·L^-1; Lane5: vitamin E; Lane6: succinate; Lane7: UT control.

Figure 4 The expression of caspase-8 protein in SGC-7901 cells fol-lowing treatment of VES for 24 h. Lane1: Molecular weight marker; Lane2: UT control; Lane3: succinate; Lane4: vitamin E; Lane5: VES at 5 mg·L^-1; Lane6: VES at 10 mg·L^-1; Lane7: VES at 20 mg·L^-1.

Figure 5 The expression of FADD protein when Fas antisense was transfected into SGC-7901 cells following treatment of VES for 24 h.