Copyright ©The Author(s) 2015.
World J Gastroenterol. Jul 7, 2015; 21(25): 7683-7708
Published online Jul 7, 2015. doi: 10.3748/wjg.v21.i25.7683
Figure 1
Figure 1 Scheme of mechanism of development of pruritus in primary biliary cirrhosis.
Figure 2
Figure 2 Xanthelasmas on both eye lids in a patient with primary biliary cirrhosis.
Figure 3
Figure 3 The scheme of the development of osteoporosis in primary biliary cirrhosis.
Figure 4
Figure 4 Stage I primary biliary cirrhosis. Inflammatory infiltrate consisting of mononuclear cells around the bile duct (BD). Hematoxylin eosin staining, magnification × 125.
Figure 5
Figure 5 Stages II-III primary biliary cirrhosis. A: Wide septa formed by proliferating ductuli. Lymphocytic infiltration. Hematoxylin eosin staining, magnification × 200. B: Hyaline inclusions. Swelling and hepatocellular injury due to the damaging effect of bile acids. van Gieson's, magnification × 200.
Figure 6
Figure 6 Stages IV primary biliary cirrhosis. Cirrhotic transformation. Silver impregnation method for Gomory, magnification × 200.
Figure 7
Figure 7 Schematically summarizes the clinical, biochemical, immunological, and morphological signs of primary biliary cirrhosis.