Hyponatremia in cirrhosis: Pathophysiology and management

doi:10.3748/wjg.v21.i11.3197

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1930

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325

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13008

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Mar 21, 2015 (publication date) through Apr 26, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Mar 21, 2015; 21(11): 3197-3205
Published online Mar 21, 2015. doi: 10.3748/wjg.v21.i11.3197

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Figure 1 Proposed mechanisms for the development of hyponatremia. SNS: Sympathetic nervous system; RAAS: Renin-angiotensin-aldosterone system; NO: Nitric oxide; RES: Reticuloendothelial system; PG: Prostaglandin; AVP: Arginine vasopressin; ADH: Antidiuretic hormone.

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Figure 2 Mechanisms involved in the splanchnic vasodilation in cirrhosis. ADH: Antidiuretic hormone.

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Figure 3 Mechanism of action of vaptans. AVP: Arginine vasopressin; AQP2: Aquaporin-2; AQP3: Aquaporin-3; AQP4: Aquaporin-4.

Citation: John S, Thuluvath PJ. Hyponatremia in cirrhosis: Pathophysiology and management. World J Gastroenterol 2015; 21(11): 3197-3205
URL: https://www.wjgnet.com/1007-9327/full/v21/i11/3197.htm
DOI: https://dx.doi.org/10.3748/wjg.v21.i11.3197