Helicobacter pylori and interleukin-8 in gastric cancer

doi:10.3748/wjg.v19.i45.8192

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Dec 7, 2013; 19(45): 8192-8202
Published online Dec 7, 2013. doi: 10.3748/wjg.v19.i45.8192

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Figure 1 Roles for interleukin-8 in tumor progression and metastasis. EMT: Epithelial-mesenchymal transition; IL-8: Interleukin-8.

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Figure 2 Scheme of signaling of Helicobacter pylori-induced interleukin-8 in gastric cancer cells. PI3-K: Phosphoinositide 3-kinase; HSP: Heat shock protein; TLR: Toll-like receptor; NOX: NADPH oxidase; NOD: Nucleotide binding and oligomerization domain; IKK: IκB kinase; ROS: Reactive oxygen species; RICK: Receptor-interacting protein serine-threonine kinase; MAPK: Mitogen-activated protein kinase; NF-κB: Nuclear factor kappa B; AP-1: Activator protein-1.

Citation: Lee KE, Khoi PN, Xia Y, Park JS, Joo YE, Kim KK, Choi SY, Jung YD. Helicobacter pylori and interleukin-8 in gastric cancer. World J Gastroenterol 2013; 19(45): 8192-8202
URL: https://www.wjgnet.com/1007-9327/full/v19/i45/8192.htm
DOI: https://dx.doi.org/10.3748/wjg.v19.i45.8192