Hepatic focal nodular hyperplasia in children: Imaging features on multi-slice computed tomography

doi:10.3748/wjg.v18.i47.7048

Advanced Search

BPG is committed to discovery and dissemination of knowledge

Home / Archive / Volume 18, Issue 47

This Article

Citation of this article

Corresponding Author of This Article

Research Domain of This Article

Article-Type of This Article

Open-Access Policy of This Article

Times Cited Counts in Google of This Article

Number of Hits and Downloads for This Article

Total Article Views (6464)

All Articles published online

The chart showing PDF series, HTML series, Figures (1-3) series, Tables (1-1) series.

Item

Count

PDF

467

HTML

3914

Figures (1-3)

267

Tables (1-1)

140

Sum=4788

Publishing Process of This Article

The chart showing Browse series, Download series.

Item

Count

Browse

932

Download

744

Sum=1676

Dec 21, 2012 (publication date) through Apr 23, 2024

Times Cited of This Article

Times Cited (13)

Journal Information of This Article

Publication Name

World Journal of Gastroenterology

ISSN

1007-9327

Publisher of This Article

Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

Brief Article

World J Gastroenterol. Dec 21, 2012; 18(47): 7048-7055
Published online Dec 21, 2012. doi: 10.3748/wjg.v18.i47.7048

Open in New Tab Full Size Figure Download Figure

Figure 1 Focal nodular hyperplasia in a 2-year-old boy. A: The lesion was hypodense on pre-contrast computed tomography scan; B: The lesion was significantly enhanced in the arterial phase with enlarged feeding arteries (arrows); C: The lesion was hyperdense in the portal vein phase with hypodense central scar (arrows) and fibrous septa; D: The lesion was isodense in the equilibrium phase, and the scar shows delayed enhanced; E: Computed tomography angiography (CTA) in the arterial phase showed that the enlarged feeding artery was distorted with a spoke-wheel shaped blood supply (arrows); F: CTA in the portal vein phase showed the draining vessels directly into the hepatic vein (arrows).

Open in New Tab Full Size Figure Download Figure

Figure 2 Focal nodular hyperplasia in a 16-year-old boy. A: The lesion was hypodense on pre-contrast computed tomography scan, and central scar with much lower density were identified (arrows); B: The lesion was significantly enhanced in the arterial phase with enlarged feeding arteries penetrating into the central scar (arrows); C: The lesion was slightly hyperdense in the portal vein phase; D: The lesion was isodense in the equilibrium phase with delayed enhancement of the central scar; E: Computed tomography angiography (CTA) in the arterial phase showed that the enlarged feeding arteries were distorted, and exhibited a spoke-wheel shaped blood supply (arrows); F: CTA in the portal vein phase showed the draining hepatic vein (arrows).

Open in New Tab Full Size Figure Download Figure

Figure 3 Focal nodular hyperplasia in a 14-year-old girl. A: The lesion was hypodense on pre-contrast computed tomography image; B, C: The lesion was significantly enhanced in the arterial phase with hypodense fibrous septa; D: The lesion was isodense in the portal vein phase; E: Immunohistochemical staining of CD34 revealed the abnormal blood vessels within fibrous septa (× 100); F: Computed tomography angiography in the arterial phase showed that a spoke-wheel shaped blood supply of the focal nodular hyperplasia (arrow).

Citation: Liu QY, Zhang WD, Lai DM, Ou-yang Y, Gao M, Lin XF. Hepatic focal nodular hyperplasia in children: Imaging features on multi-slice computed tomography. World J Gastroenterol 2012; 18(47): 7048-7055
URL: https://www.wjgnet.com/1007-9327/full/v18/i47/7048.htm
DOI: https://dx.doi.org/10.3748/wjg.v18.i47.7048