Steatosis as a co-factor in chronic liver diseases

doi:10.3748/wjg.v16.i10.1171

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Mar 14, 2010 (publication date) through Apr 19, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Mar 14, 2010; 16(10): 1171-1176
Published online Mar 14, 2010. doi: 10.3748/wjg.v16.i10.1171

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Figure 1 Hypothetical physiopathological pathways leading to insulin resistance steatosis, fibrosis, hepatocellular carcinoma (HCC), apoptosis and steatosis in hepatitis C virus (HCV) infection. NS3: Non structural HCV protein 3; Core: Core HCV protein; ROS: Reactive oxygen species; TNF-α: Tumor necrosis factor α; TGF-β: Transforming growth factor β; TG: Triglycerides; LPL: Lipo-protein-lipase; SOCS3: Signaling of cytokine suppressor type 3; IRS 1-2: Insulin receptor substrate type 1 and 2.

Citation: Persico M, Iolascon A. Steatosis as a co-factor in chronic liver diseases. World J Gastroenterol 2010; 16(10): 1171-1176
URL: https://www.wjgnet.com/1007-9327/full/v16/i10/1171.htm
DOI: https://dx.doi.org/10.3748/wjg.v16.i10.1171