Inducible nitric oxide synthase polymorphism is associated with the increased risk of differentiated gastric cancer in a Japanese population

doi:10.3748/wjg.v12.i39.6361

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Oct 21, 2006 (publication date) through Apr 18, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Oct 21, 2006; 12(39): 6361-6365
Published online Oct 21, 2006. doi: 10.3748/wjg.v12.i39.6361

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Figure 1 Regulation of Nitric oxide (NO) and polyamine pathways. H pylori induces inducible nitric oxide synthase (iNOS) expression, arginase II and ornithine decarboxylase (ODC). iNOS competes with arginase for arginine. Gastric cancer relates to the high expression of iNOS. H pylori-induced iNOS is inhibited by spermine produced by the arginase-ODC pathway.

Citation: Goto Y, Ando T, Naito M, Goto H, Hamajima N. Inducible nitric oxide synthase polymorphism is associated with the increased risk of differentiated gastric cancer in a Japanese population. World J Gastroenterol 2006; 12(39): 6361-6365
URL: https://www.wjgnet.com/1007-9327/full/v12/i39/6361.htm
DOI: https://dx.doi.org/10.3748/wjg.v12.i39.6361