Exploring the potential function of high expression of ANAPC1 in regulating ubiquitination in hepatocellular carcinoma

doi:10.4251/wjgo.v17.i5.103594

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May 15, 2025 (publication date) through Jun 5, 2025

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World Journal of Gastrointestinal Oncology

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1948-5204

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastrointest Oncol. May 15, 2025; 17(5): 103594
Published online May 15, 2025. doi: 10.4251/wjgo.v17.i5.103594

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Figure 9 Regulation of high ANAPC1 expression in cell communication, particularly in the migration inhibitory factor pathway. A: Weights/strengths of cell-cell interactions; B: Signaling roles of different cell clusters in the Midkine signaling pathway; C: Heatmap of cell types involved in the migration inhibitory factor pathway; D: Distribution of signal transduction genes participating in the Midkine signaling pathway. NK: Natural killer; MIF: Migration inhibitory factor; CXCR: C-X-C chemokine receptor; CD: Cluster of differentiation.

Citation: Tang YX, Wu WZ, Zhou SS, Zeng DT, Zheng GC, He RQ, Qin DY, Huang WY, Chen JT, Dang YW, Tang YL, Chi BT, Zhan YT, Chen G. Exploring the potential function of high expression of ANAPC1 in regulating ubiquitination in hepatocellular carcinoma. World J Gastrointest Oncol 2025; 17(5): 103594
URL: https://www.wjgnet.com/1948-5204/full/v17/i5/103594.htm
DOI: https://dx.doi.org/10.4251/wjgo.v17.i5.103594