Copyright
©The Author(s) 2021.
World J Gastroenterol. Apr 28, 2021; 27(16): 1785-1804
Published online Apr 28, 2021. doi: 10.3748/wjg.v27.i16.1785
Published online Apr 28, 2021. doi: 10.3748/wjg.v27.i16.1785
Figure 4 Knockdown of MRPL35 inhibits gastric carcinoma cell proliferation and monoclonal formation and promotes gastric carcinoma cell apoptosis.
A and B: The Celigo cell count assay was used to analyze the proliferation of AGS cells (A) and HGC-27 cells (B) transfected with shCtrl (negative control virus) and shMRPL35 (lentiviral particles of MRPL35); C and D: Colony formation assay of shCtrl and shMRPL35 infected AGS cells (C) and HGC-27 cells (D); E and F: Flow cytometry analysis of apoptosis of shCtrl and shMRPL35 infected AGS cells (E) and HGC-27 cells (F). n = 3. bP < 0.01. shCtrl: Negative control virus; shMRPL35: Lentiviral particles of MRPL35.
- Citation: Yuan L, Li JX, Yang Y, Chen Y, Ma TT, Liang S, Bu Y, Yu L, Nan Y. Depletion of MRPL35 inhibits gastric carcinoma cell proliferation by regulating downstream signaling proteins. World J Gastroenterol 2021; 27(16): 1785-1804
- URL: https://www.wjgnet.com/1007-9327/full/v27/i16/1785.htm
- DOI: https://dx.doi.org/10.3748/wjg.v27.i16.1785