Longitudinal decrease in platelet counts as a surrogate marker of liver fibrosis

doi:10.3748/wjg.v26.i38.5849

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Oct 14, 2020; 26(38): 5849-5862
Published online Oct 14, 2020. doi: 10.3748/wjg.v26.i38.5849

Table 3 Distribution of cirrhosis etiology and complications among cirrhotic patients (n = 5258)

	Frequency	Percent
Etiology
ALD	488	9.28
Viral	987	18.77
Autoimmune	159	3.02
Wilson	9	0.17
NAFLD	413	7.85
Complications
Ascites	450	8.56
Varices	551	10.48
HE	294	5.59
HCC	10	0.19
SBP	118	2.24
Portal HTN	511	9.72
Splenomgally	764	14.53
PVT	70	1.33

ALD: Alcoholic liver disease; NAFLD: Non-alcoholic fatty liver disease; HE: Hepatic encephalopathy; HCC: Hepatocellular carcinoma; SBP: Spontaneous bacterial peritonitis; HTN: Hypertension; PVT: Portal vein thrombosis.

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Citation: Gotlieb N, Schwartz N, Zelber-Sagi S, Chodick G, Shalev V, Shibolet O. Longitudinal decrease in platelet counts as a surrogate marker of liver fibrosis. World J Gastroenterol 2020; 26(38): 5849-5862
URL: https://www.wjgnet.com/1007-9327/full/v26/i38/5849.htm
DOI: https://dx.doi.org/10.3748/wjg.v26.i38.5849

Gotlieb N, Schwartz N, Zelber-Sagi S, Chodick G, Shalev V, Shibolet O. Longitudinal decrease in platelet counts as a surrogate marker of liver fibrosis. World J Gastroenterol 2020; 26(38): 5849-5862 [PMID: 33132639 DOI: 10.3748/wjg.v26.i38.5849]

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