Type I and type II Helicobacter pylori infection status and their impact on gastrin and pepsinogen level in a gastric cancer prevalent area

doi:10.3748/wjg.v26.i25.3673

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Jul 7, 2020; 26(25): 3673-3685
Published online Jul 7, 2020. doi: 10.3748/wjg.v26.i25.3673

Table 3 Distribution of CagA, VacA, UreA and UreB serological antibodies in stepwise chronic gastric disease

Groups	CagA	VacA	UreA	UreB
NAG, n = 140	89 (63.6)	82 (55.6)	93 (66.4)	138 (98.6)
NAGE, n = 75	45 (60.0)	36 (48.0)	44 (58.7)	74 (98.7)
PU, n = 85	65 (76.5)	57 (67.0)	67 (78.8)	83 (97.6)
CAG, n = 64	51 (79.7)	43 (67.2)	49 (76.6)	64 (100.0)
GC, n = 38	32 (84.2)	28 (73.7)	31 (81.6)	38 (100.0)
Total, n = 402	282 (70.1)	249 (61.9)	284 (70.6)	397 (98.8)

Data are presented as n (%). CAG: Chronic atrophic gastritis; GC: Gastric cancer; NAG: Non-atrophic gastritis; NAGE: Non-atrophic gastritis with erosion; PU: Peptic ulcer.

Citation: Yuan L, Zhao JB, Zhou YL, Qi YB, Guo QY, Zhang HH, Khan MN, Lan L, Jia CH, Zhang YR, Ding SZ. Type I and type II Helicobacter pylori infection status and their impact on gastrin and pepsinogen level in a gastric cancer prevalent area. World J Gastroenterol 2020; 26(25): 3673-3685
URL: https://www.wjgnet.com/1007-9327/full/v26/i25/3673.htm
DOI: https://dx.doi.org/10.3748/wjg.v26.i25.3673