©The Author(s) 2017.
World J Gastroenterol. May 21, 2017; 23(19): 3440-3448
Published online May 21, 2017. doi: 10.3748/wjg.v23.i19.3440
Published online May 21, 2017. doi: 10.3748/wjg.v23.i19.3440
Table 3 Summary results of proteins investigated
| Cell lines | p53 status | AXL | E-Cadherin |
| Positive control | |||
| HeLa | HPV inactivated | Highest | Negative |
| Colon | |||
| HCT116 | Wild-Type | Negative | High |
| RKO | Wild-Type | High | Negative |
| HCT116.p53 | Mutant | High | Lowest |
| RKO.p53-/- | Mutant | Highest | Negative |
| SW480 | Mutant | Highest | High |
| Breast | |||
| MCF7 | Wild-Type | Negative | Low |
| MDA-MB-361 | Wild-Type | Negative | Highest |
| ZR-75-1 | Wild-Type | Negative | Highest |
| CAL-51 | Wild-Type | Negative | Low |
| 1001 | Mutant | Highest | Negative |
| MDA-MB-231 | Mutant | High | Negative |
| T47D | Mutant | Negative | Highest |
| BT-549 | Mutant | Lowest | Negative |
- Citation: Abdel-Rahman WM, Al-khayyal NA, Nair VA, Aravind SR, Saber-Ayad M. Role of AXL in invasion and drug resistance of colon and breast cancer cells and its association with p53 alterations. World J Gastroenterol 2017; 23(19): 3440-3448
- URL: https://www.wjgnet.com/1007-9327/full/v23/i19/3440.htm
- DOI: https://dx.doi.org/10.3748/wjg.v23.i19.3440