Mechanism and pathobiologic implications of CHFR promoter methylation in gastric carcinoma

doi:10.3748/wjg.14.5000

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Aug 28, 2008 (publication date) through May 5, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

Gastric Cancer

World J Gastroenterol. Aug 28, 2008; 14(32): 5000-5007
Published online Aug 28, 2008. doi: 10.3748/wjg.14.5000

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Figure 2 Representative results of RT-PCR in human GC tissue samples. β-actin was used as an internal control. CHFR mRNA expression level in 16 and 6 poorly differentiated adenocarcinoma tissue samples and 1 mucinous adenocarcinoma tissue sample was significantly lower than that in paired normal gastric mucosa samples from the same patients, while no difference was found in 7 well-differentiated adenocarcinoma tissue samples. Marker, D2000: Marker; N: Normal gastric mucosa corresponding to tumors; T: Gastric cancer.

Citation: Gao YJ, Xin Y, Zhang JJ, Zhou J. Mechanism and pathobiologic implications of CHFR promoter methylation in gastric carcinoma. World J Gastroenterol 2008; 14(32): 5000-5007
URL: https://www.wjgnet.com/1007-9327/full/v14/i32/5000.htm
DOI: https://dx.doi.org/10.3748/wjg.14.5000