Acute liver failure: A progress report

BPG is committed to discovery and dissemination of knowledge

Home / Archive / Volume 1, Issue 1

Citation of this article

Corresponding Author of This Article

Article-Type of This Article

Open-Access Policy of This Article

Times Cited Counts in Google of This Article

Journal Information of This Article

Publication Name

World Journal of Gastroenterology

ISSN

1007-9327

Publisher of This Article

Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

Commentary

World J Gastroenterol. Oct 1, 1995; 1(1): 4-8
Published online Oct 1, 1995. doi: 10.3748/wjg.v1.i1.4

Table 4 Possible predisposing factors for acute liver failure

Etiologic agents
Viruses	various hepatotropic viruses
	superinfection of hepatotropic viruses
	variants of a hepatotropic virus (mutants)
Chemicals
Miscellaneous
Host factors
Hyperfunction of cellular immunity
Hyperfunction of antibody production → immune complexes
Endogenous endotoxemia
Deficient phagocytosis of reticuloendothetial system
Activation of macrophages	overproduction of TNF-α and IL-1
	release of leukotrienes
	release of superoxides
Liver regeneration failure	overproduction of regeneration-suppressing factors
	disorders in cell receptors and signal transduction
Apoptosis

Citation: Pan BR, Yang SF, Ma LS. Acute liver failure: A progress report. World J Gastroenterol 1995; 1(1): 4-8
URL: https://www.wjgnet.com/1007-9327/full/v1/i1/4.htm
DOI: https://dx.doi.org/10.3748/wjg.v1.i1.4

Pan BR, Yang SF, Ma LS. Acute liver failure: A progress report. World J Gastroenterol 1995; 1(1): 4-8 [DOI: 10.3748/wjg.v1.i1.4]

All content on this site: Copyright © 1993-2026 Baishideng Publishing Group Inc, its licensors, and contributors. All rights are reserved, including those for text and data mining, AI training, and similar technologies. For all open access content, the relevant licensing terms apply.