Clinical and experimental study on gastric mucosal pathology, gene expression, cAMP and trace elements of pixu patients

Figure 1 Focal superficial inflammation, partial rupture of the epithelial cytomembrane, and exposure of the cytoplasmic granule.

Figure 2 Focal intestinal metaplasia, with microvilli outside of cells, unclear intracellular granules and partial degradation of epithelial cells.

Figure 3 Focal atrophic inflammation, with a large area of cells with maintained contour but with ruptured cytomembranes and exposed cytoplasmic granules. There were only a few red blood cells present.

Figure 4 Micro-ulcer, with mucosa scaled in the ulcer area, clear granule and normal cells in the periphery. There was a small amount of red blood cells and white blood cells present.

Figure 5 Distribution histogram of DNA levels in gastric mucosa from 15 cases of benign gastric disease due to deficiency of spleen energy.

Figure 6 Distribution histogram of DNA levels in gastric mucosa from 9 cases of benign gastric disease due to spleen deficiency with energy stagnation.

Figure 7 Distribution histogram of DNA levels in gastric mucosa from 11 cases of gastric cancer.