Topic Highlight
Copyright ©2011 Baishideng Publishing Group Co.
World J Hepatol. May 27, 2011; 3(5): 114-117
Published online May 27, 2011. doi: 10.4254/wjh.v3.i5.114
Figure 1
Figure 1 Overview of the pathway of alcoholic injury resulting in the direct production of oxidative stress and the LPS-TLR pathway that culminates in production of cytokines and other inflammatory processes that result in hepatitis of the liver.
Figure 2
Figure 2 Link between gut derived bacterial lipopolysacaride and Toll like receptor-4 stimulation within the liver.