Aberrant DNA-PKcs and ERGIC1 expression may be involved in initiation of gastric cancer

doi:10.3748/wjg.v23.i33.6119

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Sep 7, 2017; 23(33): 6119-6127
Published online Sep 7, 2017. doi: 10.3748/wjg.v23.i33.6119

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Figure 1 Representative graphs of HE staining (20 ×) of various gastric tissues including dysplasia.

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Figure 2 Outline of proteomic experimental workflow. SDS-PAGE: Sodium dodecyl sulfate polyacrylamide gel electrophoresis; C-HPLC: Capillary-high performance liquid chromatography; QE: Q-exactive; LFQ: Label free quantitative.

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Figure 3 Immunohistochemical staining of DNA-PKcs (40 ×). A: Normal; B: Mild dysplasia; C: Moderate dysplasia; D: Severe dysplasia; E: Early mucosal cancer.

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Figure 4 Immunohistochemical staining of ERGIC1 (40 ×). A: Normal; B: Mild dysplasia; C: Moderate dysplasia; D: Severe dysplasia; E: Early mucosal cancer.

Citation: Wang FR, Wei YC, Han ZJ, He WT, Guan XY, Chen H, Li YM. Aberrant DNA-PKcs and ERGIC1 expression may be involved in initiation of gastric cancer. World J Gastroenterol 2017; 23(33): 6119-6127
URL: https://www.wjgnet.com/1007-9327/full/v23/i33/6119.htm
DOI: https://dx.doi.org/10.3748/wjg.v23.i33.6119