Dual protective role of HO-1 in transplanted liver grafts: A review of experimental and clinical studies

doi:10.3748/wjg.v17.i26.3101

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Jul 14, 2011 (publication date) through Apr 25, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Jul 14, 2011; 17(26): 3101-3108
Published online Jul 14, 2011. doi: 10.3748/wjg.v17.i26.3101

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Figure 1 Function of heme oxygenase 1 and its degradation product in ischemia and reperfusion injury during liver transplantation. HO-1: Heme oxygenase 1; CO: Carbon monoxide; SEC: Sinusoidal endothelial cell; ROS: Reactive oxygen species; IRI: Ischemia-reperfusion injury.

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Figure 2 Function of heme oxygenase 1 degradation product to reduce rejection. CO: Carbon monoxide; Treg: Regulatory T cells; IL: Interleukin.

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Figure 3 Perfusion of donor liver with heme oxygenase 1 inducers could reduce rejections through many pathways. HO-1: Heme oxygenase 1; DC: Dendritic cell; Treg: Regulatory T cells; IDO: Indoleamine 2,3-dioxygenase; MC: Mast cell.

Citation: Wang CF, Wang ZY, Li JY. Dual protective role of HO-1 in transplanted liver grafts: A review of experimental and clinical studies. World J Gastroenterol 2011; 17(26): 3101-3108
URL: https://www.wjgnet.com/1007-9327/full/v17/i26/3101.htm
DOI: https://dx.doi.org/10.3748/wjg.v17.i26.3101