Energetic etiologies of acute pancreatitis: A report of five cases

doi:10.4291/wjgp.v6.i4.243

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World Journal of Gastrointestinal Pathophysiology

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2150-5330

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastrointest Pathophysiol. Nov 15, 2015; 6(4): 243-248
Published online Nov 15, 2015. doi: 10.4291/wjgp.v6.i4.243

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Figure 1 Schematic model illustrating possible mechanisms of neurogenic pancreatitis. AP: Acute pancreatitis; STT: Spinothalamic tract; TENS: Transcutaneous electrical nerve stimulation; DRG: Dorsal root ganglion; IL: Interleukin; TNF: Tumor necrosis factor; NK-1R: Neurokinin receptor 1; 1oSN: Primary sensory neuron; NG: Nodose ganglion; Pg: Prostaglandins; SP: Substance P; TRPV-1: Transient receptor potential vanilloid 1; CGRP: Calcitonin gene related protein; NkA: Neurokinin A; IP3R: Inositol-3-phosphate receptor; RyR: Ryanodine receptor.

Citation: Shmelev A, Abdo A, Sachdev S, Shah U, Kowdley GC, Cunningham SC. Energetic etiologies of acute pancreatitis: A report of five cases. World J Gastrointest Pathophysiol 2015; 6(4): 243-248
URL: https://www.wjgnet.com/2150-5330/full/v6/i4/243.htm
DOI: https://dx.doi.org/10.4291/wjgp.v6.i4.243