Update on molecular pathogenesis of Helicobacter pylori-induced gastric cancer

doi:10.4291/wjgp.v16.i2.107052

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Jun 22, 2025 (publication date) through Jun 19, 2025

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World Journal of Gastrointestinal Pathophysiology

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2150-5330

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastrointest Pathophysiol. Jun 22, 2025; 16(2): 107052
Published online Jun 22, 2025. doi: 10.4291/wjgp.v16.i2.107052

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Figure 1 The key virulence factors involved in the pathogenicity of Helicobacter pylori infection. BabA: Blood group antigen-binding adhesion A; SabA: Sialic acid-binding adhesion A; NAP: Neutrophil activating protein; Hsp: Heat shock protein; VacA: Vacuolating cytotoxin gene A; CagA: Cytotoxin-associated gene A; T4SS: Type IV secretion system; ROS: Reactive oxygen species; IL: Interleukin; MIP: Macrophage inflammatory protein.

Citation: Raza Y, Mubarak M, Memon MY, Alsulaimi MS. Update on molecular pathogenesis of Helicobacter pylori-induced gastric cancer. World J Gastrointest Pathophysiol 2025; 16(2): 107052
URL: https://www.wjgnet.com/2150-5330/full/v16/i2/107052.htm
DOI: https://dx.doi.org/10.4291/wjgp.v16.i2.107052