Residual risk in atherosclerotic cardiovascular disease after statin therapy: Clinical mechanisms and management strategies

doi:10.4330/wjc.v18.i2.114960

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World Journal of Cardiology

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1949-8462

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Cardiol. Feb 26, 2026; 18(2): 114960
Published online Feb 26, 2026. doi: 10.4330/wjc.v18.i2.114960

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Figure 2 Mechanistic overview of residual cardiovascular risk (low-density lipoprotein-independent lipid and inflammatory pathways). TRLs: Triglyceride-rich lipoproteins; LPL: Lipoprotein lipase; eNOS: Endothelial nitric oxide synthase; LDL: Low-density lipoprotein; Lp(a): Lipoprotein(a); HDLC: High-density lipoprotein cholesterol; Apo B: Apolipoprotein B; IL: Interleukin; TNF: Tumor necrosis factor; hsCRP: High-sensitivity C-reactive protein; LDL-C: Low-density lipoprotein-cholesterol.

Citation: Tan SH, Wu JL, Zhuo SX, Zhang Y, Wang M. Residual risk in atherosclerotic cardiovascular disease after statin therapy: Clinical mechanisms and management strategies. World J Cardiol 2026; 18(2): 114960
URL: https://www.wjgnet.com/1949-8462/full/v18/i2/114960.htm
DOI: https://dx.doi.org/10.4330/wjc.v18.i2.114960

Tan SH, Wu JL, Zhuo SX, Zhang Y, Wang M. Residual risk in atherosclerotic cardiovascular disease after statin therapy: Clinical mechanisms and management strategies. World J Cardiol 2026; 18(2): 114960 [PMID: 41694036 DOI: 10.4330/wjc.v18.i2.114960]

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