Myocardial reperfusion injury and oxidative stress: Therapeutic opportunities

doi:10.4330/wjc.v10.i9.74

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Sep 26, 2018 (publication date) through Apr 25, 2024

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World Journal of Cardiology

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1949-8462

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Cardiol. Sep 26, 2018; 10(9): 74-86
Published online Sep 26, 2018. doi: 10.4330/wjc.v10.i9.74

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Figure 4 Experimental, pharmacological and clinical approaches to prevent myocardial reperfusion injury at cellular level. RISK: Reperfusion injury salvage kinase pathway; SAFE: Survivor activating factor enhancement pathway; GSH: Reduced glutathione; GSSG: Oxidized glutathione; NAC: N-acetylcysteine; DFO: deferoxamine; ONOO*: Peroxynitrite; NO: Nitric oxide; OH *: Radical hydroxyl; mPTP: Mitochondrial permeability transition pore.

Citation: González-Montero J, Brito R, Gajardo AI, Rodrigo R. Myocardial reperfusion injury and oxidative stress: Therapeutic opportunities. World J Cardiol 2018; 10(9): 74-86
URL: https://www.wjgnet.com/1949-8462/full/v10/i9/74.htm
DOI: https://dx.doi.org/10.4330/wjc.v10.i9.74