Letter to the Editor: Bile acids in cholangiocarcinoma: Dual regulation via the hippo-yes-associated protein pathway and model limitations

doi:10.4251/wjgo.v18.i6.116624

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World Journal of Gastrointestinal Oncology

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1948-5204

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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Copyright: ©Author(s) 2026. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution-NonCommercial (CC BY-NC 4.0) license. No commercial re-use. See permissions. Published by Baishideng Publishing Group Inc.

World J Gastrointest Oncol. Jun 15, 2026; 18(6): 116624
Published online Jun 15, 2026. doi: 10.4251/wjgo.v18.i6.116624

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Figure 2 Primary bile acids are synthesized in the liver from cholesterol via the classical or alternative pathway. Upon entering the intestine, they are metabolized by gut microbiota into secondary bile acids. Approximately 90%-95% of these are reabsorbed in the terminal ileum and recycled by the liver through enterohepatic circulation. BA: Bile acid; CA: Cholic acid; GCA: Glycocholic acid; TCA: Taurocholic acid; CDCA: Chenodeoxycholic acid; GDCA: Glycodeoxycholic acid; TDCA: Taurodeoxycholic acid; DCA: Deoxycholic acid; UDCA: Ursodeoxycholic acid; GUDCA: Glycoursodeoxycholic acid; TUDCA: Tauroursodeoxycholic acid; LCA: Lithocholic acid; GLCA: Glycolithocholic acid; TLCA: Taurolithocholic acid.

Citation: Wu KL, Guo D, Yang L, Li YQ, Wu YX, Zhou CF, Tang JF. Letter to the Editor: Bile acids in cholangiocarcinoma: Dual regulation via the hippo-yes-associated protein pathway and model limitations. World J Gastrointest Oncol 2026; 18(6): 116624
URL: https://www.wjgnet.com/1948-5204/full/v18/i6/116624.htm
DOI: https://dx.doi.org/10.4251/wjgo.v18.i6.116624

Wu KL, Guo D, Yang L, Li YQ, Wu YX, Zhou CF, Tang JF. Letter to the Editor: Bile acids in cholangiocarcinoma: Dual regulation via the hippo-yes-associated protein pathway and model limitations. World J Gastrointest Oncol 2026; 18(6): 116624 [DOI: 10.4251/wjgo.v18.i6.116624]

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