FNDC5 enhances quercetin-induced anoikis in pancreatic adenocarcinoma cells via focal adhesion kinase-dependent mechanisms

doi:10.4251/wjgo.v18.i5.118849

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World Journal of Gastrointestinal Oncology

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1948-5204

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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Copyright: ©Author(s) 2026. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution-NonCommercial (CC BY-NC 4.0) license. No commercial re-use. See permissions. Published by Baishideng Publishing Group Inc.

World J Gastrointest Oncol. May 15, 2026; 18(5): 118849
Published online May 15, 2026. doi: 10.4251/wjgo.v18.i5.118849

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Figure 5 Diagram illustrating the underlying mechanism that FNDC5 enhanced the quercetin-induced anoikis in pancreatic ductal adenocarcinoma cell via FNDC5/irisin/focal adhesion kinase/phosphatidylinositol 3-kinase/protein kinase B axis. ECM: Extracellular matrix; FAK: Focal adhesion kinase; AKT: Protein kinase B; PI3K: Phosphatidylinositol 3-kinase; NF-κB: Nuclear factor kappa-B.

Citation: Gao HF, Zhang K, Cheng CS, Shen YH, Chen H. FNDC5 enhances quercetin-induced anoikis in pancreatic adenocarcinoma cells via focal adhesion kinase-dependent mechanisms. World J Gastrointest Oncol 2026; 18(5): 118849
URL: https://www.wjgnet.com/1948-5204/full/v18/i5/118849.htm
DOI: https://dx.doi.org/10.4251/wjgo.v18.i5.118849

Gao HF, Zhang K, Cheng CS, Shen YH, Chen H. FNDC5 enhances quercetin-induced anoikis in pancreatic adenocarcinoma cells via focal adhesion kinase-dependent mechanisms. World J Gastrointest Oncol 2026; 18(5): 118849 [DOI: 10.4251/wjgo.v18.i5.118849]

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