Increasing expression of presenilin 1, β-catenin, and p-PTEN and its regulatory roles on cell invasion in gastric cancer

Figure 1 Prognostic values of presenilin-1, β-catenin, and phosphorylation of tensin homolog deleted on chromosome ten in gastric cancer tissues. A: Representative immunohistochemical staining of presenilin-1 (PS-1), β-catenin, and phosphorylation of tensin homolog deleted on chromosome ten (p-PTEN) in continuous tissue sections from gastric tumor tissues. Original magnification, × 40; B: Significant differences of metastatic lymph nodes between low and high expression of PS-1 (left), β-catenin (middle), and p-PTEN (right); C: The overall survival rate of patients with high PS-1 (left), β-catenin (middle), and p-PTEN (right) expression were significantly lower than that of patients with low protein expression. PS-1: Presenilin-1; p-PTEN: Phosphorylation of tensin homolog deleted on chromosome ten.