PARVA promotes gastric cancer progression through FBXO15-related PI3K/AKT signaling

doi:10.4251/wjgo.121827

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World Journal of Gastrointestinal Oncology

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1948-5204

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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Copyright: ©Author(s) 2026. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution-NonCommercial (CC BY-NC 4.0) license. No commercial re-use. See permissions. Published by Baishideng Publishing Group Inc.

World J Gastrointest Oncol. Oct 15, 2026; 18(10): 121827
Published online Oct 15, 2026. doi: 10.4251/wjgo.121827

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Figure 6 FBXO15 partially reversed the inhibitory effects of PARVA knockdown on malignant phenotypes of gastric cancer cells. A: Colony formation assays with quantification; B: EdU staining images after FBXO15 overexpression in PARVA-silenced cells; C: Quantification of EdU-positive cells; D: Transwell migration and invasion assays with quantification; E: Wound-healing assays with quantification of wound closure. ^aP < 0.001. NS: Not significant; Pa: PARVA; Fb: FBXO15; OE: Overexpression.

Citation: Lu X, Li J, Wang ML, Wang HZ, Li YX. PARVA promotes gastric cancer progression through FBXO15-related PI3K/AKT signaling. World J Gastrointest Oncol 2026; 18(10): 121827
URL: https://www.wjgnet.com/1948-5204/full/v18/i10/121827.htm
DOI: https://dx.doi.org/10.4251/wjgo.121827

Lu X, Li J, Wang ML, Wang HZ, Li YX. PARVA promotes gastric cancer progression through FBXO15-related PI3K/AKT signaling. World J Gastrointest Oncol 2026; 18(10): 121827 [DOI: 10.4251/wjgo.121827]

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