©The Author(s) 2025.
World J Gastrointest Oncol. Sep 15, 2025; 17(9): 108892
Published online Sep 15, 2025. doi: 10.4251/wjgo.v17.i9.108892
Published online Sep 15, 2025. doi: 10.4251/wjgo.v17.i9.108892
Figure 7 F4-enhanced cytotoxic T lymphocyte induced the apoptosis of CT26 cells.
A: The proportions of dendritic cells in the tumor tissues, which were detected by flow cytometry; B: The proportions of cluster of differentiation (CD) 8+ T cells in the tumor tissues, and the interferon-γ expression in the CD8+ T cells, which were detected by flow cytometry; C: The expressions of apoptosis-related proteins in the tumor tissues of CT26-bearing mice. All data were shown as mean ± SD (n = 5). aP < 0.05. P vs model group. MHC: Major histocompatibility complex; Ctrl: Control; CD: Cluster of differentiation; CAP: Capecitabine; L: Low; M: Middle; H: High; LPS: Lipopolysaccharides; SSC: Side scatter; IFN: Interferon.
- Citation: Xie W, Li XJ, Zhong YS, Fang J, Qi H, Yang M, Ying HZ, Yu CH. Ginsenoside F4 inhibits colorectal cancer progression by boosting dendritic cell maturation and remodeling the tumor microenvironment. World J Gastrointest Oncol 2025; 17(9): 108892
- URL: https://www.wjgnet.com/1948-5204/full/v17/i9/108892.htm
- DOI: https://dx.doi.org/10.4251/wjgo.v17.i9.108892