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©The Author(s) 2025.
World J Gastrointest Oncol. Jun 15, 2025; 17(6): 105782
Published online Jun 15, 2025. doi: 10.4251/wjgo.v17.i6.105782
Published online Jun 15, 2025. doi: 10.4251/wjgo.v17.i6.105782
Table 3 Binding affinity of active ingredients to the hub targets
| Ingredient | Target | ||
| ESR1 | STAT3 | TP53 | |
| 1-2-Di-O-linolenoylglycerol-3-O-β-D-galactopyranoside | -6.3 | -5.5 | -5.6 |
| Anethole | -5.6 | -4.4 | -5.2 |
| β-sitosterol | -6.9 | -5.9 | -6.4 |
| Chlorogenic acid | -7.8 | -6.7 | -7.1 |
| Daucosterol | -6.7 | -7.2 | -6.7 |
| Isoquercitrin | -7.7 | -6.8 | -6.7 |
| Isovitexin-2-O-rhamnoside | -5.4 | -7 | -8.2 |
| Phytol | -7 | -4.3 | -5.3 |
| Roseoside | -7.4 | -6.2 | -6.9 |
| Vitrxin-2-O-rhamnoside | -1.3 | -6.4 | -7.7 |
- Citation: Dong YD, Wu XM, Liu WQ, Hu YW, Zhang H, Fang WD, Luo Q. Potential mechanism of Camellia luteoflora against colon adenocarcinoma: An integration of network pharmacology and molecular docking. World J Gastrointest Oncol 2025; 17(6): 105782
- URL: https://www.wjgnet.com/1948-5204/full/v17/i6/105782.htm
- DOI: https://dx.doi.org/10.4251/wjgo.v17.i6.105782