Molecular mechanism of nanomaterials induced liver injury: A review

doi:10.4254/wjh.v16.i4.566

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Apr 27, 2024 (publication date) through May 18, 2024

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World Journal of Hepatology

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1948-5182

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Hepatol. Apr 27, 2024; 16(4): 566-600
Published online Apr 27, 2024. doi: 10.4254/wjh.v16.i4.566

Table 9 Effects and molecular mechanisms underlying Cr₂O₃NPs induced hepatonanotoxicity

NPs	Size	Tested model	Dose & route of administration	Effects & mechanism	Ref.
Cr₂O₃-NPs	22.50 + 1.76 nm (TEM)	Wistar rats	50 mg/100 g bwt (LD), 200 mg/100 g bwt (HD); single dose for 1, 7, 14 d (oral)	ALT, AST, ALP, γGT, total bilirubin (increased)	[23]

ALP: Alanine phosphatase, ALT: Alanine aminotransferase, AST: Aspartate aminotransferase, HD: High dose, LD: Low dose, γGT: Gamma glutamyltransferas.

Citation: Das SK, Sen K, Ghosh B, Ghosh N, Sinha K, Sil PC. Molecular mechanism of nanomaterials induced liver injury: A review. World J Hepatol 2024; 16(4): 566-600
URL: https://www.wjgnet.com/1948-5182/full/v16/i4/566.htm
DOI: https://dx.doi.org/10.4254/wjh.v16.i4.566