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©The Author(s) 2021.
World J Hepatol. Nov 27, 2021; 13(11): 1552-1567
Published online Nov 27, 2021. doi: 10.4254/wjh.v13.i11.1552
Figure 1
Figure 1 Pathophysiology of Wilson’s disease. Due to mutation in ATP 7B gene, P type ATPase is defective and copper is not incorporated in ceruloplasmin. Free copper increases in blood and is deposited in liver and extrahepatic sites (brain, kidneys, bones, cornea, RBC).


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