ENaCδ aggravates gastroesophageal reflux disease via PI3K/AKT/mTOR: Human umbilical cord mesenchymal stem cells restore barrier function

doi:10.4252/wjsc.120457

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World Journal of Stem Cells

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1948-0210

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Copyright: ©Author(s) 2026. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution-NonCommercial (CC BY-NC 4.0) license. No commercial re-use. See permissions. Published by Baishideng Publishing Group Inc.

World J Stem Cells. Jun 26, 2026; 18(6): 120457
Published online Jun 26, 2026. doi: 10.4252/wjsc.120457

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Figure 1 Pathology of mouse esophageal tissue (hematoxylin and eosin staining, × 200). C: Wild-type C57BL/6 mic; hENaCδ: Humanized δ subunit of epithelial sodium channel; GERD: Gastroesophageal reflux disease; PPI: Proton pump inhibitor; hUC-MSCs: Human umbilical cord mesenchymal stem cells.

Citation: Wang C, Li Y, Chen QQ, Wan J. ENaCδ aggravates gastroesophageal reflux disease via PI3K/AKT/mTOR: Human umbilical cord mesenchymal stem cells restore barrier function. World J Stem Cells 2026; 18(6): 120457
URL: https://www.wjgnet.com/1948-0210/full/v18/i6/120457.htm
DOI: https://dx.doi.org/10.4252/wjsc.120457

Wang C, Li Y, Chen QQ, Wan J. ENaCδ aggravates gastroesophageal reflux disease via PI3K/AKT/mTOR: Human umbilical cord mesenchymal stem cells restore barrier function. World J Stem Cells 2026; 18(6): 120457 [DOI: 10.4252/wjsc.120457]

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